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Fig. 6 | Genes and Environment

Fig. 6

From: Exploring the role of oxidative stress and mitochondrial dysfunction in β-damascone-induced aneuploidy

Fig. 6

Effect of N-acetyl-l-cysteine (NAC) on mitochondrial membrane potential in BEAS-2B cells exposed to β-damascone (β-Dam) or mitochondrial poison. The mitochondrial membrane potential fold changes were expressed as the average fluorescence ratio of polarized and depolarized after 4 h of exposure to β-Dam (a) or a mitochondrial poison, carbonyl cyanide 4-(trifluoromethoxy) phenylhydrazone (FCCP) (b). These values were calculated from the solvent control value without NAC co-treatment. The solid and dotted lines represent the values obtained with or without NAC co-treatment, respectively. Each result represents the mean and standard error of the mean for three independent experiments. #Significantly different from the solvent control group (P < 0.05, Dunnett's test)

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